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By B. Sandstedt (auth.), E. Grundmann (eds.)

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The problem could perhaps be settled with in situ angiography of the uterus and placenta. The significance of extrachorial implantation for the LBW syndrome has already been discussed. The few but large cotyledons in extrachorial placentas imply that mere largeness of cotyledons cannot be associated with better intrauterine fetal growth as suggested by Crawford (1965a, b) and Schordania (1929). , a local uterine factor. Because of its deeper but less extensive manner of growth, it is possible that PE makes contact with fewer spiral arteries.

II. Neurological and intellectual sequelae. : White infarcts of the placenta. Br. J. Obstet. Gynaecol. : The pattern of villous variability in the normal placenta. Br. J. Obstet. Gynaecol. : Pevillious fibrin deposition in the human placenta. Am. J. Obstet. Gynecol. : Placenta extrachorialis. A clinico-pathological study. Br. J. Obstet. Gynaecol. : Die Feinstruktur der Chorionzotten bei Splitgestose unterschiedlicher Schweregrade. Z. Geburtshilfe Gynaekol. : A contribution to clarification of structure and function of the placenta.

Winick (1967) showed that there is no further increase in cell numbers after approximately the 36th week of gestation. The elongated slender types of growing ends, which are frequent in early pregnancy, are not present (/vemark and Sandstedt, 1971; Bergstrom, 1971). b) Trophoblastic Layer Erythrocytes are occasionally present on the syncytial surface. They seem to adhere but are not enclosed in depressions or microvilli. Apart from cut and fractured leSions, the syncytial surface is intact. On the fractured surface, the trophoblast forms a homogeneous layer of varying thickness covering the villous core (see Figs.

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