Download Neuroimmunology of Sleep by S. R. Pandi-Perumal PDF

By S. R. Pandi-Perumal

This well timed paintings displays the turning out to be value of study into the character of sleep and its scientific elements. one of many first of its variety, this paintings at the neuroimmunology of sleep presents an advent to the interaction among those key and interlinked parts. Written either from a easy and a scientific point of view, the amount is a useful details source for biomedical pros and scholars of the human biology.
Highly useful advisor written from either a simple and medical point of view.

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Wrona, D. (2006) Neural-immune interactions: An integrative view of the bidirectional relationship between the brain and immune systems. J Neuroimmunol 172, 38–58. , and Pawlikowski, M. (1989) Effect of growth hormone-releasing hormone on human peripheral blood leukocyte chemotaxis and migration in normal subjects. Neuroendocrinology 50, 236–239. 2 Neuroimmunological Correlates of Circadian Rhythmicity in Oral Biology and Medicine Francesco Chiappelli, Olivia S. Cajulis, Audrey Navarro, and David R.

1995) Differential cellular regulation of POMC by IL-1 and corticotrophin-releasing hormone. Neuroendocrinology 61, 136. , and Kawamura, N. (2002) Coemergence of insomnia and a shift in the Th1/Th2 balance toward Th2 dominance. Neuroimmunomodulation 10, 337–343. M. (2003) Chronic insomnia and immune functioning. Psychosom Med 65, 211–221. M. (1995) Soluble interleukin-6 (IL-6) receptor augments central effects of IL-6 in vivo. FASEB J 9, 659. , and Stepien, H. (2005) Effect of growth hormone-releasing hormone [GHRH(1-44)NH2] on IL-6 and IL-8 secretion from human peripheral blood mononuclear cells in vitro.

IFN-Ȗ at physiological concentrations has been shown to inhibit stimulated secretion of ACTH, PRL, and GH of pituitary cells cultured in vitro and stimulated with hypothalamic factors (Vankelecom et al. 1990). These results indicate that IFN-Ȗ may modulate GH secretion from the pituitary gland by both a direct suppressive effect at the level of the pituitary and indirect hypothalamic suppression involving stimulation of SOM release (Gonzalez, Aguila, and McCann 1991). TNF-Į is a cytokine produced by activated macrophages or monocytes that is important in the hormonal response to shock (Kelley, Arkins, and Li 1992).

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