By Stefan Silbernagl, Florian Lang
pathophysiology is step one towards scientific medication. This
book offers an important mechanisms of ways disease
develops in approximately two hundred brilliant colour plates observed by way of transparent text.
Every double web page, with textual content at the left and photographs at the right,
gives a swift and entire review of its topic.
are prepared systematically and a color-coded thumb index makes it
easy to navigate in the course of the booklet. the place invaluable for readability, the
basics of body structure are defined again.
references illustrate the hyperlinks among thought and perform. The pocket
atlas has been totally revised and up-to-date for the 3rd version. It now
includes precise information regarding malaria, power metabolism, eating
disorders, and weight problems. Color Atlas of Pathophysiology is the precise complement to Color Atlas of Physiology!
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Extra resources for Color Atlas of Pathophysiology
IL-10) are also responsible for limiting the rise in fever ( to ca. 41°C). Fever-reducing drugs (antipyretics ) are effective here. For example, acetylsalicylic acid inhibits cyclo- oxygenases 1 and 2, which catalyze the production of PGE2 from arachidonic acid. v. injection of lipopolysaccharides ( components of the cell wall of gram-negative bacteria) the above-mentioned cytokines are released only 30 minutes after the onset of the fever and their appearance can be inhibited by subdiaphragmatic vagotomy, it seems that exogenous pyrogens also activate the preoptic area and the VOLT via afferent fibers from the abdomen.
A small amount of Hb released intravascularly is bound to haptoglobin (-> p. 40 ). In massive acute intravascular hemolysis (-> B ) haptoglobin is, however, overloaded and free Hb is filtered in the kidneys (-» p. 44 ). This results not only in hemoglobinuria ( dark urine ), but can also lead to acute renal failure through tubular occlusion (-» p. 118 ). Chronic hemoglo binuria additionally causes Fe deficiency anemia , cardiac output rises and the resulting mechanical hemolysis creates a vicious circle ( -» B ).
A. Heat Collapse, Heatstroke Core temperature Normothermic ? 2 Heat stroke Plate -> Death CNS damage B. Malignant Hyperthermia Inhalation anesthesia, muscle relaxants If genetically disposed / Sarcoplasmic reticulum @ @ Generalized muscle contractions Dantrolene \ Lactic acid \ 02 deficiency \ ^\l \ \ \ \ \ \ Heat production ff Cellular loss of K + Acidosis Vasodilation — Energy consumption ff r Arrhythmias, heart failure \ Drop in blood pressure ^ Hyperkalemia > Core temperature ft Coma <- 27 Hypothermia, Cold Injury Energy , Tempratue 2 If there is a danger of the core temperature dropping, (counter)regulatory heat production results ( muscle tremor and movement) (-> A).